NFKB1 mRNA is the target of miR-9, and this leads to the loss of translation of NF-κB; however, this outcome is avoided by the action of OC43, which binds to miR-9, allowing NF-κB translation, pro-inflammatory cytokine production, and type I interferon production, which are necessary to resolve the infection [69]. The gene discussed is NFKB1; the disease is infection.