Identificationof proteins, such as syntaxin-1A and the chemokine KC/GRO, highlightstheir potential role in Alzheimer’s disease, opening up newpossibilities to further study their association with Aβ pathology.We also saw that mitochondrial and neuronal developmental proteinsthat are frequently altered in Alzheimer’s disease were shiftedby the SST-scFv8D3 treatment towards normal levels. This evidence concerns the gene STX1A and early-onset autosomal dominant Alzheimer disease.