CD38 and asthma: These two processes are probably both driven by the same stimulation, as the type‐I IFN signalling‐induced expression of these antiviral proteins also plays an important role during B‐cell differentiation,9, 10, 11 antibody class‐switching12 and antibody secretion.9, 13 Similarly, an elevated expression of antiviral genes was also accompanied by increased expression of antibody genes and plasmablast markers CD27, CD38 and PRDM1 in asthma patients on day 3 after infection.