Expression of a constitutively active form of the latter (referred to as GPA-12*) recapitulates many of the transcriptional changes that accompany infection [5], including an increased expression of nlp-29 (Fig 3D), and also leads to a high level of expression of the nlp-29p::GFP reporter, making this a useful tool for epistasis analysis [46]. Here, NINL is linked to infection.