None of the strains induced significant differences in the expression of OAS1 after rotavirus infection, while both L. salivarius strains increased the expressions of PKR, Mx1 and RNAseL. L. salivarius FFIG35 was more efficient for increasing RNAseL while the FFIG58 strain was more efficient for the up-regulation of PKR (Figure 6). The gene discussed is EIF2AK2; the disease is Rotavirus infection.