IL33 and infection: IL-33 is produced by pulmonary epithelial and endothelial cells, AM and natural killer T (NKT) cells following infection with A/WSN/1933 H1N1, as well as A/Memphis/71 H3N2 IAV and in response, ILC2 proliferate and can exacerbate airway hyperreactivity in an IL-13-dependent manner in the absence of T cells [86,87,204].