Relevant pathways related to pulmonary fibrosis included “ECM remodeling” and “TGFβ-induced fibroblast/myofibroblast migration and extracellular matrix production in asthmatic airways.” Immune response related pathways included “Classical complement pathway,” “Lectin induced complement pathway,” “Common mechanisms of Th17 cell migration,” and “T cell co-signaling receptors” (Figure 4B). The gene discussed is TGFB1; the disease is pulmonary fibrosis.