KAT2A, a histone acetyltransferase (HAT) and a member of the GCN5-related N-acetyltransferase (GNAT) superfamily, interacts with acetyl-CoA and transfers its acetyl residues to histones (Grant et al., 1997; Wang and Dent, 2014) and is associated with gene expression, tumor formation, and tumor cell proliferation (Wang et al., 2017). The gene discussed is GLYATL1; the disease is neoplasm.