CAMK2G and myocardial infarction: During MI, CaMKII phosphorylation leads to the phosphorylation of Nav1.5, a cardiac sodium channel (Howard et al., 2018), and the reverse of Ito (a cardiac potassium channel) by decreasing Kv4.3 gene (Tao et al., 2020; Tinaquero et al., 2020), inducing arrhythmias.