LILRB2 and neoplasm: In addition, there was a relatively high level of interaction between tumor cells and TAMs, particularly via the MHC I-LILRB axis, where MHC class I molecules (B2M and HLA) and LILRB molecules (LILRB1 and LILRB2) were expressed in the respective cells for immunosuppression that supports tumor growth in other cancers18,19 (Supplementary Fig. 16).