Consistently, in our data, markers of cardiac (troponin T, NT-proBNP) and renal (creatinine, urea) function, as well as anemia and dyserythropoiesis (hemoglobin, hematocrit, erythrocytes, and red blood cell distribution width) correlate with various APPs (APOA2, APOE, CD14, GSN, LYZ, SAA1, SAA2, and SERPINA3; Figure 1B and Table S4) supporting the role of inflammation in COVID-19-related organ damage and its impact on erythropoiesis. This evidence concerns the gene LYZ and anemia (phenotype).