Patients with AAA have high levels of systemic inflammatory acute-phase reactants, such as hs-C-reactive protein, IL-1β, IL-6, interferon-γ, tumor necrosis factor-α, α-1-antitrypsin, orosomucoid, haptoglobin, and fibrinogen (5, 29–31). The gene discussed is TNF; the disease is triple-A syndrome.