On the contrary, Jae Won Chang et al. used P11, an inhibitor of PAFAH1b3, to block this enzyme making a dramatically decrease on the survival of plentiful mouse and human cancer cell lines cultivated under serum-starved conditions, which displayed that the protumorigenic function of PAFAH1b3 relies on its activity, rather than just expression (21). The gene discussed is PAFAH1B3; the disease is cancer.