SEs and BET proteins such as BRD4 contribute to inflammatory or malignant processes and activate translocated oncogenes.7, 8 Widespread development of BET inhibitors notably includes treatment of solid tumors to reduce post‐radiotherapy lung fibrosis,8, 9, 10, 11 and these drugs have rapidly entered into therapy trials, eventually ahead of science.12 This evidence concerns the gene DNER and pulmonary fibrosis.