In the process of IPF, mitochondria activate the TGFβ1 pathway after excessive stress during oxidative stress, and NOX4 is overexpressed in NADPH oxidase, which further promotes the process of pulmonary fibrosis [20–23], which may be one of the most critical processes in the process of IPF, which may be effectively reversed by MitoQ. This evidence concerns the gene FMO5 and idiopathic interstitial pneumonia.