ATP2A2 and myocardial infarction: Interestingly, the latter presented an underlying mechanism we identified ourselves during transcriptome analysis in mice: the mentioned up-regulation of Atp2a2 in MstnLn/Ln mice with concurrent down-regulation in WT mice, which was paralleled by up-regulation of SERCA2a (translational product of Atp2a2) in the setting of cardioprotection after myocardial infarction in mice47.