Chronic myeloid leukemia (CML), which is characterized by the presence of the Philadelphia chromosome (Ph +) and the resultant BCR-ABL1 fusion, occurs in different phases dependent on the absence or presence of certain clinical features, including cytogenetic clonal evolution, basophilia, and elevated blast percentage [1]. Here, ABL1 is linked to chronic myelogenous leukemia, BCR-ABL1 positive.