To investigate whether two other candidate genes (PIGP and DSCR3) are involved in the pathological features of DS, we transfected piggyBac transposon vectors containing human PIGP and/or DSCR3 cDNA under regulation of the Tet-inducible system into NPCs differentiated from Partial-Tri21 iPSCs. This evidence concerns the gene VPS26C and Dravet syndrome.