Overall, these observations suggest that elevated FGF10 levels in BESCs may induce efficient epithelial repair via BESCs surrogacy for incapacitated AT2 progenitors in case of severe lung injury (Figure 2A) and that FGF10-based therapies based on this phenomenon may prove beneficial for ARDS and/or COVID-19 patients. The gene discussed is FGF10; the disease is acute respiratory distress syndrome.