The hypothesis of the CRC tumor origin of 26:0, 28:0 and 26:1 VLCFAs in the serum of CRC patients is also supported by our earlier study that showed the high overexpression of elongases ELOVL1 and ELOVL6 (elongation of very long chain fatty acids protein 1 and 6) in CRC tumors, which are responsible for the elongation of SFAs and MUFAs (6). This evidence concerns the gene ELOVL1 and neoplasm.