Previous studies have reported that the levels of reactive oxygen species (ROS), C-reactive protein (CRP), and proinflammatory cytokines were significantly increased when endothelial dysfunction occurs, which led to the enhanced expression and secretion of some adhesion molecules, such as intercellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1), and E-, L-, and P-selections, and consequently promoting the concentration of blood and the formation of small patches [6, 7]. The gene discussed is VCAM1; the disease is endothelial dysfunction.