Furthermore, Wang et al. (2017) identified three TE indels, Alu-5072, Alu-5075, and SVA-282, in the class II region as potential enhancers for HLA-DRB5, HLA-DQB1-AS1, and HLA-DPB2 associated with GWAS phenotypes of lymphoma, Hodgkin lymphoma, and chronic hepatitis B infection, respectively. This evidence concerns the gene HLA-DPB2 and chronic hepatitis B virus infection.