Infection with A2-O significantly induced the expression of SA-regulated genes that encode basic pathogenesis-related protein 1 (PR1) and β-1,3-glucanase (PR2), as well as a JA biosynthesis gene (lipoxygenase D (LoxD)), the JA-regulated gene protease inhibitor II (PI-II), and two ET biosynthesis genes (1-aminocyclopropane-1-carboxylate synthase (ACS) and 1-aminocyclopropane-1-carboxylate oxidase (ACO)) (Farmer et al., 1992; Yan et al., 2013; Booker and DeLong, 2015; Zhang et al., 2018; Marhavý et al., 2019; Figure 2C). Here, KLK15 is linked to infection.