CD68 and autoimmune myocarditis: Though we found no sex differences in the number of immune reactive CD68+ macrophages in our EAM model, hearts from female EAM rats were infiltrated with an increased amount of anti-inflammatory CD68+ ArgI+ macrophages, suggesting that a predominant phenotype in females is alternative activated macrophages (M2) that favor an anti-inflammatory environment thus attenuating inflammation in female hearts in autoimmune myocarditis.