CD4 and hepatitis C virus infection: However, the pathways that were observed at higher FDR (>5%) included adaptive immune response, CD4-positivity, alpha-beta T-cell stimulation, methyl-branched fatty acid metabolic process, fatty acid biosynthesis, B-cell receptor signaling pathway, Hepatitis C and metabolic pathways which seem biologically plausible in the context of our study since these pathways are related to chronic inflammation.