WES and the subsequent bioinformatics analysis revealed that both the cell line and the donor tumor tissues had a nonsynonymous mutation in exon 7 of TP53 (TP53G245S), which is reportedly a hotspot mutation in TP53 (variant allele frequency: 8.3% and 49.3% in the donor tumor tissue and the cell culture, respectively) [23]. This evidence concerns the gene TP53 and neoplasm.