Indeed, LSEC from ImmTOR-treated animals were capable of suppressing proliferation of stimulated T cells and they (as well as KC) also expressed much lower amounts of neutrophil chemokine KC/GRO (Figure 7), which is known to play a key role in several models of inflammatory response (24, 25, 52) and hepatic production of which has been shown to correlate with hepatic infiltration by neutrophils in a model of experimental sepsis (53). The gene discussed is CXCL1; the disease is Sepsis.