FABP1 and Hepatic fibrosis: On the contrary, resolution from CCl4‐induced fibrosis was impaired in mice with hepatocyte‐ and HSCs‐ L‐FABP deletion but not altered in mice with either hepatocyte‐ or HSC‐alone L‐FABP deletion.[56] In the in vitro study, L‐FABP was found to decrease in concomitant with the loss of lipids during HSC activation while its overexpression restored lipid formation and inhibited HSC activation.[55, 57 These findings implicated the beneficial effect of L‐FABP in the resolution of liver fibrosis by restoring quiescence in HSC.