A‐FABP is part of a finely tuned positive feedback loop with JNK and AP‐1 to exacerbate LPS‐induced inflammatory responses in macrophages.[32] Thus, the possibility was explored that A‐FABP induces TGFβ1 transactivation in HSCs in liver fibrosis through potentiating JNK/c‐Jun signaling. This evidence concerns the gene JUN and Hepatic fibrosis.