Taken together with the evidence that HSCs predominately secrete TIMPs contributing to liver fibrosis[47] whereas pharmacological inhibition of A‐FABP attenuates the hepatic expression of TIMP‐1 in diet‐induced obese mice,[10] this evidence support the detrimental action of A‐FABP in liver fibrosis is at least partially exerted through its regulation on TGFβ1 expression. Here, TGFB1 is linked to Hepatic fibrosis.