Of the genes more highly expressed in female FLT3‐ITD‐positive AML, the hedgehog signalling mediator GLI2 has been associated with FLT3‐ITD‐mutated leukaemia [42], while HOXB‐AS3 is reportedly upregulated in NPM1‐mutated AML, and has been implied to contribute in regulation of AML cell‐cycle progression [43]. The gene discussed is PDS5B; the disease is acute myeloid leukemia.