A pronounced innate immune response to infection followed the peak of viral replication, evidenced by induction of cytokines (IL‐6, TNF), chemokines (CCL2, CCL5) and type I and III IFNs (IFNβ, IFNλ1/3) measured by RT–qPCR (Figs 1F and G, and EV2D–F). This evidence concerns the gene IFNL1 and infection.