The majority of ALS patients, regardless of genetic status, show cytoplasmic TDP‐43 positive protein aggregates in motor neurons, implicating this protein in ALS pathology (Neumann et al., 2006), while rare mutations in its gene, TARDBP, are found in some patients (Zou et al., 2017). This evidence concerns the gene TARDBP and amyotrophic lateral sclerosis.