STAT3 and retinoblastoma: LncRNA SNHG14 was reported to be function as a competing endogenous RNA (ceRNA) of miR-124, upregulating signal transducer and activator of transcription 3 (STAT3); consequently, SNHG14 silencing inhibited cell proliferation, migration and invasion as well as promoted apoptosis in retinoblastoma cells (45).