found that the expression of PCED1B-AS1 was downregulated in patients with active TB, and PCED1B-AS1 acted as an endogenous sponge to block the expression of miR-155 in macrophages by directly binding to miR-155, thereby reducing the apoptosis rate and promoting autophagy (Figures 2B and 4) (106). The gene discussed is PCED1B; the disease is tuberculosis.