In support of this hypothesis, tandem genomic duplications in metastatic prostate cancers were found in regions harbouring an intergenic enhancer element upstream of the androgen receptor gene (AR) and near the MYC gene, both loci‐encoding transcription factors that play a crucial oncogenic role in prostate cancer and whose overexpression has been associated with dysregulated transcription and RNA‐processing [158, 159]. The gene discussed is MYC; the disease is Familial prostate cancer.