FOXP3 and myocardial infarction: As the number of anti‐inflammatory CD4+ Foxp3+ Tregs between CD4+ T cell population in heart‐draining lymph nodes is known to be elevated in the wake of MI,51 it can be postulated that DC‐EXs likely activate cardioprotective Tregs after infarction and, thereby, beneficially impact wound healing and survival.