Following MI, Tregs infiltrate into the myocardium and induce the differentiation of M2 macrophages and highly secrete anti‐inflammatory factors, like IL‐10 and TGF‐β, to inhibit the inflammatory response of M1 macrophages and lymphocytes within the healing myocardium, thereby ameliorating the inflammation‐mediated cardiac damage and improving wound healing.100, 103. Here, IL10 is linked to myocardial infarction.