Considering that miR-375/SLC7A11 axis may play a role through other forms of cell death instead of inducing ferroptosis, GC cells with miR-375 overexpression or SLC7A11 knockdown were treated with Fer-1 (10 μM), apoptosis inhibitor Z-VAD-FMK (20 μM) [53], or necrosis inhibitor Nec-1 (0.1 mM) [54, 55], and following by examining the stemness of GC cells. This evidence concerns the gene SLC7A11 and gastric cancer.