Furthermore, we found that dormant residual tumor cells in vivo exhibit increased expression of Tgfbr3 (TGFβR-III), as well as its ligand Tgfb2 (TGFβ-II), and markedly decreased expression of Plaur (uPar) compared to both primary and recurrent tumor cells (Fig. 4c). The gene discussed is TGFBR3; the disease is neoplasm.