Additionally, after inhibiting miR-29c-3p in BM-MSCs, Wnt3a and β-catenin levels were noticeably reduced in EVs-treated AD neurons (all p < 0.001) (Figure 7A, 7B), which were then obviously restored after BACE1 knockdown (all p < 0.01) (Figure 7A, 7B). This evidence concerns the gene BACE1 and Alzheimer disease.