In addition, other genes were enriched in processes confirmed to be also strongly associated with the pathogenesis and progression of T2DM such as the “MAPK signaling,” “EGFR signaling,” and “T cell receptor signaling pathway.” The findings suggest that m6A methylation factors may be modulating the expression of genes in pancreatic β-cells of patients with T2DM. This evidence concerns the gene EGFR and type 2 diabetes mellitus.