In nude mice with LSD1 mutant-expressing MDA-MB-231 tumors, ectopic overexpression of wild-type LSD1 or a phosphorylation-mimicking LSD1-S111D (originally reported as S112 referring to mouse LSD1, while S111 is the human LSD1 residue) enhanced metastasis, whereas overexpression of a phosphorylation-defective S112A mutant did not change the metastatic potential of cancer cells [50]. Here, KDM1A is linked to cancer.