Our observation of the increase in the BM percentages of GMPs and monocytes suggested that the leukemic cell mass was, at least in part, directed to the monocytic precursors dysregulated by Gata2-L359V mutation on the basis of BCR/ABL-induced CML (30.3% vs 18.5%). This evidence concerns the gene BCR and chronic myelogenous leukemia, BCR-ABL1 positive.