ILC3 produces IL-17 and IL-22, mobilizes neutrophils to defend against extracellular bacteria and fungi, and is involved in epithelial cell activation and proliferation, and in the liver, prevents the development of nonalcoholic fatty liver disease (NAFLD) and nonalcoholic steatohepatitis (NASH) [16]. Here, IL17A is linked to metabolic dysfunction-associated steatohepatitis.