Major improvements that might be hypothesized are (i) lumping together MMRd carcinomas, regardless of grade and histotype (with the exception of SWI/SNF-deficient UEC/DEC) and (ii) substratifying the risk in NSMP EECs based on further prognostic factors (e.g., CTNNB1 mutations). Here, CTNNB1 is linked to carcinoma.