These studies reported that the differential expressed genes were associated with various pathways including (1) fibrosis [23]; (2) cell adhesion, focal adhesion, cardiac muscle contraction [54]; (3) T cell receptor signaling pathway, primary immunodeficiency, endometrial cancer, drug metabolism cytochrome P450, tyrosine metabolism, complement and coagulation cascades, and Jak-STAT signaling pathway [36]; (4) mitochondrial oxidative signaling and noncanonical autophagy [46]. This evidence concerns the gene SOAT1 and inborn error of immunity.