To date, it is known that UCA-1 shows association with HIF-1α in hypoxic environments [213], participates in the regulation of EMT processes though targeting miR-145–ZEB1/2 or miR-143/HMGB1 pathways [15,214,215], induces cisplatin/gemcitabine xenobiotic metabolism modulating miR-196a [216], and, curiously, it is under the regulation of miR-1 [217], one of the miRNAs involved in metabolic reprogramming in BC. This evidence concerns the gene UCA1 and breast cancer.