Furthermore, both also highlighted in IPF BALF an up-regulation of inflammatory mediators, such as macrophage migration inhibitory factor (MIF) and calgranulin B (S100A9) [33,34,35], which were further evaluated and confirmed to be more abundant in BALF of IPF patients by Bargagli et al. [37,38] and Hara et al. [39]. This evidence concerns the gene S100A9 and idiopathic pulmonary fibrosis.