Hong et al. [21] found that hsa-miR-320a-3p was involved in the regulation of EMT, functioning by suppressing the level of E-cadherin and upregulating expression of N-cadherin and vimentin by directly targeting forkhead box M1 (FOXM1). In general, there are two ways that miR-320 negatively controls EMT to play its tumor-suppressive role. This evidence concerns the gene FOXM1 and neoplasm.