EGR3 and glioblastoma: They also found that overexpression of hsa-miR-320b by binding to EGR3 inhibited the expression of p-PI3K and p-Akt, suggesting that hsa-miR-320b was related to the EGR3-induced modulation of glioblastoma cell malignant progression that occurs via the EGFR/PKP2 pathway.