Hira et al. affirmed that inhibition of semaphorin 3A (Sema3A) in the subacute phase of stroke suppressed astrocyte activation and negatively regulated miR-30c-2-3p and miR-326-5p in As-Exos to promote axonal outgrowth and functional recovery in MCAO rats by increasing prostaglandin D2 synthase [65]. The gene discussed is SEMA3A; the disease is stroke disorder.