Additional data analyses revealed that the main biological function outcomes of OC-X treatments could potentially be attributed to the MYC downregulation by a more than −2.4-fold change, plausibly suppressing all previously reported TNBC progression stages, including neoplasia, cell proliferation, cell-to-cell signaling and interaction, invasion, migration, and metastasis (Figure 7B). Here, MYC is linked to neoplasm.