Vitamin B1 plays an important role in intracellular glucose metabolism by acting as a coenzyme for α-ketoglutarate dehydrogenase complexes, transketolase, and pyruvate dehydrogenase [36], and our study shows that vitamin B1 intakes were significantly lower and that levels of HbA1c and fasting glucose were significantly higher in individuals with MetS. The gene discussed is TKT; the disease is metabolic syndrome.